Tool

Self-assessment

Twenty-four single-best-answer questions across anatomy, physiology, technique, interpretation and disease patterns. Three study modes: Browse filters by level & topic; Spaced uses a five-box Leitner schedule with 10 min, 1, 3, 7 and 21 day intervals; Streak graduates a card after three consecutive corrects; Timed picks a fresh random ten.

24 questions

Why is the head tilted 30° forward at the start of a rotational chair test?

Which structure has a 'baseline' time constant of approximately 4–6 s?

Which frequencies are typical for a SHA protocol?

A patient shows reduced gain at every SHA frequency and a step-test Tc of 5 s with intact visual suppression. The most likely diagnosis is:

Phase lead is increased and asymmetry is high one week after an acute right-sided neuritis. Six months later, SHA looks normal. The best interpretation is:

A patient has a step-test Tc of 35 s, normal gain, and impaired visual suppression of nystagmus. The lesion most likely involves:

Between attacks of Ménière's disease, the most likely SHA finding is:

A SHA frequency returns a spectral purity of 42 %. The correct action is:

Slow-phase eye velocity during VOR is in which direction relative to chair velocity?

A 42-year-old with sound-induced vertigo has loud-AC oVEMP amplitudes and a low cVEMP threshold. SHA is normal. The most consistent diagnosis is:

An acute right-sided horizontal nystagmus with reduced right vHIT gain, reduced caloric response on the right, and a normal posterior-canal vHIT. The likely site is:

Inter-ictal SHA in vestibular migraine most commonly shows:

Which cranial nerve carries vestibular afferent fibres centrally?

Which of these patterns most strongly suggests a central rather than peripheral lesion?

Why might a vestibular schwannoma sometimes produce only mild RCT findings despite a large tumour?

Per the 2017 Bárány criteria, a SHA-based diagnosis of bilateral vestibulopathy requires a gain below which threshold across frequencies?

Vision is denied during SHA recording chiefly because:

The earliest age-related RCT change is typically:

What does the rotational chair test offer that the caloric test does not?

VOR gain at 0.32 Hz in a healthy adult is typically:

Six months after a left labyrinthectomy a patient's SHA is symmetric and within normal limits, but the caloric test still shows a 90 % left weakness. The best explanation is:

Which nucleus is the principal site of the velocity-storage integrator?

Which of the following best distinguishes uncompensated unilateral peripheral loss from a central lesion on RCT?

Why does the step-velocity test use both an acceleration and a deceleration phase?