Treatment · Introduction

Pharmacological management of vertigo

Two jobs, one rule. Drugs in vertigo either suppress the acute storm or treat the underlying disease — and the suppressants, however useful, must be stopped before they get in the way of recovery.

Two kinds of drug

Trainee

The first group is the vestibular suppressants — antihistamines, anticholinergics, benzodiazepines and dopamine antagonists — which dampen vestibular signalling for short-term comfort but do nothing for the cause.4 The second is disease-targeted therapy aimed at specific mechanisms: betahistine and diuretics for hydrops, corticosteroids for neuritis, migraine prophylaxis, and SSRIs for PPPD.1

The one rule worth memorising

Vestibular suppressants relieve symptoms by damping vestibular input — but that same damping blunts vestibular compensation, the neuroplastic recalibration that actually restores balance. So they are for the first 3–5 days of an acute episode and no longer; then they come off and rehabilitation begins.3

suppressantsdays 0–5compensation + rehabilitationif suppressants continuedtime →function →
Vestibular suppressants relieve the acute crisis but blunt the neuroplastic recalibration that drives recovery. The teaching rule is to use them for the first 3–5 days only, then stop and start vestibular rehabilitation.

How this chapter is organised

  • Vestibular suppressants — antihistamines, anticholinergics, benzodiazepines and dopamine antagonists, with their receptor targets and the 3–5 day rule.
  • Disease-targeted therapy — Ménière’s disease, vestibular neuritis, vestibular migraine and PPPD.
  • Central vertigo — treating the underlying stroke, MS or tumour, with suppressants in a supporting role only.
  • Special populations & cautions — duration of use, the elderly, and pregnancy.
  • Emerging therapies — neuromodulation, NMDA and cannabinoid modulation, and inner-ear drug delivery.
  • Drug formulary — the full, filterable reference of every agent in the chapter.

Key points

  • Vertigo drugs are either symptomatic suppressants or disease-targeted therapy.
  • Suppressants give short-term relief but do not treat the cause.
  • Limit suppressants to 3–5 days — longer use delays vestibular compensation.
  • Targeted therapy (betahistine, steroids, migraine prophylaxis, SSRIs) addresses specific mechanisms.
  • Pharmacology supports, but does not replace, repositioning manoeuvres and rehabilitation.