The disorder
Clinical features
A patient who is suddenly, continuously spinning for days, veering to one side, with a steady beat of the eyes — and, in pure neuritis, hearing that is entirely normal.
The cardinal features
The attack starts over hours and does not stop — it is there even lying still, for days. The patient feels sick, struggles to walk and tends to fall to one side, and their eyes flick steadily in one direction. In neuritis the hearing is normal.
Vestibular neuritis is the acute vestibular syndrome: acute, sustained vertigo with nausea, gait imbalance and a unidirectional spontaneous nystagmus.1 The 2022 Bárány criteria require acute/subacute sustained spinning, a peripheral spontaneous nystagmus and an ipsilesional head-impulse deficit, with central signs excluded.3 Tap each feature:
Three features carry the diagnosis at the bedside: the direction of the nystagmus (away from the lesion, obeying Alexander’s law), the side of the head-impulse deficit and the fall (toward the lesion), and the absence of central signs. The single most reassuring observation is a nystagmus that is suppressed by fixation and an abnormal head impulse on the side the patient falls toward.2
The peripheral nystagmus
The nystagmus is horizontal-torsional and unidirectional, beating away from the affected ear. Its peripheral signature is that visual fixation suppresses it — remove fixation (Frenzel goggles or the dark) and it grows. Toggle fixation below:
For the full taxonomy of nystagmus and its localising value, see the Nystagmus chapter; for how it is recorded, see VNG.
The course of recovery
The acute crisis settles over days; the static deficit then compensates over weeks, and the dynamic deficit recovers over months. Step through the trajectory:
Severe continuous vertigo, nausea and vomiting, prostrate with spontaneous nystagmus. Symptomatic relief and short-course steroids belong here; bed rest beyond a day or two is counter-productive.
Around half of patients report some residual imbalance.2 Two sequelae deserve active follow-up: a secondary BPPV in the spared posterior canal, and persistent postural-perceptual dizziness when symptoms outlast the deficit — see BPPV and the planned PPPD chapter.
Key points
- The acute vestibular syndrome: acute, continuous vertigo lasting days, present at rest.
- Unidirectional horizontal-torsional nystagmus beating away from the lesion, suppressed by fixation.
- Gait veers and the head impulse is abnormal toward the affected side; hearing is spared in neuritis.
- Recovery runs acute → subacute compensation → chronic; watch for secondary BPPV and PPPD.