Module · Glossary
Glossary
37 terms covering the language of the vertigo history — from acute vestibular syndrome to the SO STONED mnemonic. Each definition links to related terms and, where applicable, to the relevant section of the chapter. Bookmark terms to revisit; search by term, alias, or any word in a definition.
A
Acute vestibular syndrome
AVSSudden, continuous vertigo lasting more than 24 hours with nausea, vomiting, spontaneous nystagmus, and an unsteady gait. The key split is benign (vestibular neuritis) versus dangerous (cerebellar or brainstem stroke), resolved at the bedside by HINTS.
AICA
anterior inferior cerebellar arteryThe anterior inferior cerebellar artery supplies the inner ear as well as the lateral pons and cerebellum. An AICA infarct can mimic peripheral vertigo because the labyrinth is involved — bedside hearing testing (HINTS Plus) helps unmask it.
B
Benign paroxysmal positional vertigo
BPPVA peripheral disorder in which dislodged otoconia enter a semicircular canal, producing brief episodes of vertigo triggered by specific head movements such as lying down or rolling over in bed.
Benign paroxysmal vertigo of childhood
BPVCBrief, recurrent, spontaneous episodes of vertigo in young children who are otherwise well between attacks. Considered a migraine precursor and one of the commonest causes of childhood vertigo.
C
Central vertigo
vertigo of central originVertigo arising from the brainstem, cerebellum, or central vestibular pathways. It is typically accompanied by additional neurological deficits such as ataxia, diplopia, or dysarthria, and does not usually affect hearing.
Cerebellar stroke
An ischaemic or haemorrhagic insult to the cerebellum, often presenting with vertigo, imbalance, and nystagmus. It is frequently missed on early imaging, which is why bedside signs and a careful history matter so much.
D
Diplopia
double visionDouble vision. During vertigo it is a red flag for central pathology, particularly when combined with other cranial-nerve signs.
Disequilibrium
A sense of unsteadiness or imbalance without the illusion of spinning. It usually reflects cerebellar disease, sensory ataxia, or multisensory decline rather than an acute labyrinthine lesion.
Dysarthria
slurred speechSlurred or slowed speech from impaired motor control of articulation. In a vertiginous patient it points to brainstem involvement.
E
Endolymphatic hydrops
Distension of the endolymphatic space of the inner ear — the pathological hallmark associated with Ménière's disease, linked to vertigo, fluctuating hearing loss, tinnitus, and aural fullness.
F
Functional dizziness
Dizziness without an identifiable structural lesion, including persistent postural-perceptual dizziness (PPPD). Often associated with anxiety, hypervigilance, and sensory mismatch, and real and disabling for the patient.
H
HINTS examination
Head Impulse, Nystagmus, Test of SkewA three-step bedside oculomotor examination used to separate central from peripheral causes of the acute vestibular syndrome. A normal head impulse, direction-changing nystagmus, or a skew deviation each suggest a central cause.
HINTS Plus
HINTS with the addition of a bedside hearing check. New unilateral hearing loss raises concern for an AICA-territory infarct that can otherwise masquerade as a peripheral lesion.
L
Labyrinthitis
Inflammation affecting both the cochlear and vestibular parts of the labyrinth, producing acute vertigo together with sensorineural hearing loss and tinnitus — distinguishing it from vestibular neuritis, which spares hearing.
M
Ménière's disease
An inner-ear disorder with recurrent attacks of vertigo lasting 20 minutes to several hours, accompanied by fluctuating low-frequency hearing loss, tinnitus, and a sense of aural fullness.
Motion sensitivity
visual vertigoExaggerated dizziness provoked by complex or moving visual stimuli — scrolling screens, supermarket aisles, busy patterns. Common in vestibular migraine and PPPD.
N
Nystagmus
Involuntary rhythmic oscillation of the eyes. Its direction, whether it changes with gaze, and whether it suppresses with visual fixation help separate peripheral from central origins.
O
Oscillopsia
An illusory to-and-fro movement of the visual scene, classically with head movement or walking. A hallmark of bilateral vestibular hypofunction.
Otoconia
canalithsotolithsCalcium-carbonate crystals from the utricle. When displaced into a semicircular canal they make it gravity-sensitive, producing the brief positional vertigo of BPPV.
P
Perilymph fistula
An abnormal communication that allows perilymph to leak from the inner ear, often after barotrauma or straining. Produces pressure- and sound-induced vertigo with fluctuating hearing.
Persistent postural-perceptual dizziness
PPPDA chronic functional disorder of non-spinning dizziness or unsteadiness lasting more than three months, worse on standing, with movement, and in visually complex environments. Often follows an acute vestibular insult.
Presbyvestibulopathy
Age-related decline in vestibular function, producing chronic imbalance and unsteadiness — typically bilateral, progressive, and without acute vertigo or hearing loss.
Presyncope
A sensation of impending faint, usually cardiovascular in origin (orthostatic hypotension, arrhythmia). It is commonly mislabelled as dizziness but is not true vertigo.
Proprioceptive dysfunction
Impaired joint-position sense and spatial orientation, contributing to imbalance and disequilibrium — particularly in older adults and those with peripheral neuropathy.
R
Ramsay Hunt syndrome
herpes zoster oticusReactivation of varicella-zoster virus affecting the facial and vestibulocochlear nerves, presenting with vertigo, hearing loss, facial palsy, and a vesicular rash in the ear canal.
Red flags
Clinical features that signal a potentially serious or central cause of vertigo: focal neurological deficits, vertical or direction-changing nystagmus, acute hearing loss with facial palsy, or symptoms refractory to treatment. Any of these mandates urgent imaging and referral.
S
SO STONED mnemonic
A structured history-taking framework for vertigo: Symptoms, Onset, Speed of onset, Triggers, Otological symptoms, Neurological symptoms, Evolution, and Duration.
Spontaneous nystagmus
Nystagmus occurring without a positional or motion trigger. Its direction and its modulation by visual fixation assist in localising the lesion.
Superior canal dehiscence
SCDSCDSAbsence of bone over the superior semicircular canal creates a mobile 'third window', producing sound- and pressure-induced vertigo (the Tullio phenomenon) and autophony.
T
Tullio phenomenon
Vertigo or oscillopsia provoked by loud sound. It signals a third-window lesion such as superior canal dehiscence or perilymph fistula.
V
Vertigo
A false sensation of self- or environmental motion, typically rotational, resulting from dysfunction of the peripheral or central vestibular system. It is a symptom, not a diagnosis.
Vestibular hypofunction (bilateral)
bilateral vestibulopathyLoss of vestibular function in both labyrinths, typically presenting with chronic imbalance, oscillopsia, and difficulty walking in the dark or on uneven ground rather than acute spinning vertigo.
Vestibular migraine
migrainous vertigoA subtype of migraine causing episodic vertigo, sometimes without headache, often with photophobia, phonophobia, and a personal or family history of migraine. A leading cause of episodic vertigo in young to middle-aged adults.
Vestibular neuritis
A peripheral disorder caused by inflammation of the vestibular nerve, producing acute, continuous vertigo without hearing loss, usually self-limiting over days as central compensation occurs.
Vestibular rehabilitation therapy
VRTA specialised form of physical therapy that promotes central compensation for vestibular deficits through habituation, adaptation, and substitution exercises.
Vestibular schwannoma
acoustic neuromaA benign tumour of the vestibular nerve in the cerebellopontine angle. It typically presents with progressive asymmetric hearing loss and tinnitus, with relatively mild imbalance rather than acute vertigo.
Visual dependence
Excessive reliance on visual input for spatial orientation when vestibular or proprioceptive feedback is impaired. Common in PPPD and a driver of visually-induced dizziness.