Making the diagnosis

Differential diagnosis

The complaint — chronic unsteadiness, worse in the dark — is shared by several disorders. Bilaterally reduced vestibular testing with oscillopsia is what sets this one apart.

What else causes chronic imbalance

Trainee

Key separators: a unilateral deficit is asymmetric (spinning at onset, one positive head impulse). A cerebellar/central disorder adds gaze-evoked or downbeat nystagmus, abnormal pursuit and saccades, and limb ataxia. A sensory neuropathy gives a markedly positive Romberg with reduced vibration sense but normal vestibular tests. Functional dizziness (PPPD) has normal testing and no oscillopsia.1

Bilateral vestibulopathy against its mimics

Bilateral vestibulopathy is the reference row; tap a mimic to surface the discriminator.

ConditionTriggerHearing
Bilateral vestibulopathyreferenceChronic, progressiveOften spared (cause-dependent)
Acute then compensatedVariable
Chronic, progressiveSpared
ChronicSpared
Often post-acute eventSpared

Tap a mimic to reveal the discriminator. Bilaterally reduced testing with oscillopsia is the key.

Spinning vertigo with a single positive head impulse points to a unilateral vestibulopathy; gaze-evoked or downbeat nystagmus and limb ataxia to a central/cerebellar disorder; persistent dizziness with normal testing to PPPD. Where ataxia and a neuropathy accompany the vestibular loss, think CANVAS and pursue the genetic work-up.

Key points

  • Unilateral loss is asymmetric (spinning, one positive head impulse) — not bilateral failure.
  • Cerebellar/central disease adds central oculomotor signs and limb ataxia; sensory ataxia gives a positive Romberg with normal vestibular tests.
  • Functional dizziness (PPPD) has normal vestibular testing and no oscillopsia.
  • CANVAS combines BVP, cerebellar ataxia and neuropathy — central signs may point to a cause, not away from BVP.