The disorder

Clinical features

A patient who gets dizzy at a concert, hears their eyeballs move, and finds their own voice booming in one ear — a constellation odd enough to be diagnostic once you recognise it.

Two clusters, one cause

Trainee

The vestibular cluster is sound/pressure-induced vertigo and oscillopsia with chronic imbalance; the auditory cluster is autophony, bone-conduction hyperacusis, pulsatile tinnitus and a pseudo-conductive loss.1 Symptom mix varies — some present mainly vestibular, others mainly auditory.

Sound/pressure vertigo. Vertigo and oscillopsia provoked by loud sound (Tullio phenomenon) or by pressure — Valsalva, tragal pressure, straining (Hennebert sign). The evoked eye movements align with the plane of the superior canal (vertical-torsional).

Provoking the signs at the bedside

Because the third window responds to sound and pressure, you can unmask it. The evoked eye movements align with the plane of the superior canal (vertical-torsional). Explore the provocations:

ManoeuvrePresent a loud tone (≈ 110 dB) to the affected ear.

ResponseVertigo and oscillopsia with vertical-torsional nystagmus in the plane of the superior canal — the eyes move as if the canal were excited.

The eye movements are best seen with fixation removed — see how nystagmus is recorded and classified in the Nystagmus and VNG chapters.

Key points

  • Vestibular cluster: sound/pressure-induced vertigo and oscillopsia (Tullio, Hennebert), chronic imbalance.
  • Auditory cluster: autophony, bone-conduction hyperacusis (hearing eye movements/footsteps/pulse), pulsatile tinnitus.
  • Provoked eye movements align with the superior canal’s plane (vertical-torsional).
  • Autophony of bodily sounds and supranormal bone conduction point to a third window, not the middle ear.