Modality

Magnetic Resonance Imaging

MRI is the modality of choice whenever the bedside cannot exclude a central or retrocochlear cause. Its strength is soft-tissue contrast across the brainstem, cerebellum, cranial nerves and inner ear; its weakness is access, cost, and contraindications such as cardiac devices.

When MRI is the right answer

Trainee

The acute vestibular syndrome workhorse is DWIwith the corresponding ADC map. Restricted diffusion confirms acute infarction; reading the two together filters out the T2-shine-through false positives.1Beware the documented false-negative rate of early DWI for small posterior fossa strokes — a single negative scan in the first 24–48 h does not exclude infarction.2,3

For the IAC and cerebellopontine angle, FIESTA / CISSthin-slice T2 sequences delineate the seventh and eighth cranial nerves against bright CSF. Gadolinium-enhanced T1 then highlights schwannomas, meningiomas and active demyelinating plaques. For demyelination across the brain and cord, 3D-FLAIR is the standard, with post-Gd T1 to find active lesions.4

Endolymphatic hydrops can now be imaged directly. Delayed 3D-FLAIR acquired 4 hours after intravenous (or 24 hours after intratympanic) gadolinium resolves the perilymphatic from the endolymphatic compartment, allowing grade-based reporting of saccular and cochlear hydrops.5

The sequences — at a glance

The figure below pairs each sequence with its tissue read-out and the clinical question it best answers. The slice illustrations are schematic — not derived from any patient — and intended to clarify the contrast logic rather than substitute for radiological training.

acute infarctDWI + ADC

Schematic axial cut — not derived from a real patient.

Contrast

Acute cytotoxic oedema bright on DWI, dark on ADC

Clinical question

Acute cerebellar / brainstem infarction; the cornerstone of acute stroke imaging.

Tissue read-out

  • Normal brainmid
  • Acute infarctbright
  • T2 shine-throughbright
  • Old infarctdark

Same lesion, four sequences

The same finding does not look the same in every sequence. Pick a lesion and see which of DWI, FLAIR, post-Gd T1 and FIESTA actually reveals it — the green tick marks the sequence that earns its place in the protocol.

DWI
FLAIR
Post-Gd T1
FIESTA

Reading guide

Vestibular schwannoma

Post-Gd T1 is the diagnostic sequence (avid enhancement in the IAC). FIESTA shows the filling defect against bright CSF. DWI and FLAIR are usually unhelpful.

The internal auditory canal — what we are looking at

Four nerves run together through the IAC: facial (VII), cochlear, superior vestibular and inferior vestibular. Schwannomas almost always arise from the superior vestibular nerve; they fill the canal first, then bulge into the cerebellopontine angle. Drag the stage slider to watch the growth pattern that produces the classical “ice-cream cone.”

Normal anatomyIntracanalicular tumourCPA extension · ice-cream cone
IAC · cross-sectionBill's barfalciform crestFacialCochlearSuperior vestibularInferior vestibularCoronal · IAC + CPAbrainstemIACCoronal MRI

Schematic — not derived from a real patient scan. The vestibular schwannoma classically arises from the superior vestibular nerve's Schwann cells. Growth fills the IAC first, then bulges into the CPA cistern; the resulting profile is the “ice-cream cone.”

MRI cautions and contraindications

  • Cardiac devices and ferromagnetic implants — non-MR-conditional pacemakers and certain cochlear implants preclude MRI; choose CT alternatives or MR-conditional protocols when essential.
  • Gadolinium— modern macrocyclic agents are safe in most patients; severe renal impairment (eGFR < 30) and prior reactions require careful weighing.
  • Claustrophobia, agitation, vomiting — limit early-acquisition quality; consider sedation or short-protocol stroke MRI in the unwell patient.
  • Time and access — out-of-hours MRI access is the practical bottleneck in most centres; a clear protocol minimises wasted acquisitions.