At the bedside

Differential diagnosis

BPPV is usually obvious, but positional vertigo has impostors — some benign, one dangerous. The episode duration, the triggers, the presence of cochlear symptoms, and the behaviour of the nystagmus separate them.

1s10s1m10m1h1dBPPVVestibularmigraineMénière'sVestibularneuritisCentralpositional
Episode duration is the first discriminator. BPPV is measured in seconds; Ménière’s and migraine in minutes to hours; neuritis is continuous over days. Central positional vertigo is variable or persistent and carries red flags — pair a suspicious duration with non-fatiguing nystagmus or neurological signs and image rather than reposition.
Distinguishing BPPV from its mimics
DiagnosisEpisode durationTriggerCochlear symptomsKey clue
BPPV< 1 minPositional (lying, rolling, looking up)NoneLatency + fatigability; canal-specific nystagmus on positional testing
Vestibular migraineMinutes–hoursVariable, sometimes positionalUsually noneMigraine features (headache, photophobia, aura); no reproducible fatigable nystagmus
Ménière's disease20 min–hoursSpontaneousFluctuating low-frequency loss, tinnitus, fullnessCochlear symptoms with the vertigo
Vestibular neuritisDays (continuous)Not positionalNoneAcute prolonged vertigo; abnormal head impulse; unidirectional spontaneous nystagmus
Central positional vertigoVariable / persistentPositionalNoneNo latency, non-fatiguing, pure downbeat or direction-changing; neurological signs

The benign mimics

Vestibular migraine can be positional, but episodes run minutes to hours and usually carry migrainous features; its positional vertigo lacks the reproducible, fatigable nystagmus of BPPV. Ménière’s disease is set apart by its cochlear triad (fluctuating hearing loss, tinnitus, fullness) and longer episodes — though BPPV can coexist with it. Vestibular neuritis causes acute, continuous vertigo over days with an abnormal head impulse and is not positional; BPPV may follow it in recovery.

The dangerous mimic — central positional vertigo

Cerebellar and brainstem lesions — infarction, demyelination, tumour — can mimic positional vertigo. The distinguishing features are mechanical opposites of BPPV: nystagmus with no latency, that does not fatigue, often pure downbeat or direction-changing, and frequently lacking the torsional component of posterior-canal BPPV.1,2

Red flags that mandate MRI (diffusion-weighted) and referral: atypical or non-fatiguing nystagmus; persistent imbalance; associated cranial-nerve, cerebellar or long-tract signs (diplopia, dysarthria, limb ataxia, facial numbness); and failure to respond to a correctly performed repositioning manoeuvre.3